Fat Burners

A very common question one of the most common of all; what are the best fat loss steroids? While in truth we can accurately say almost all anabolic steroids can lead to a reduction in body fat there are agents that are definitely the best fat loss steroids of all. As there are steroids that are more apt to fulfill this purpose there are also a host of other commonly used agents often mistaken for anabolic steroids when in truth they fall into different categories. While these other agents do in fact lead to body fat reduction and may indeed be placed in the performance enhancing category, many of them are not members of the steroidal family.

The Best Fat Loss Steroids:

As it pertains to pure body fat reduction if we were to list the absolute best fat loss steroids the list would undoubtedly begin with Trenbolone. Trenbolone as a powerful muscle building, strength increasing and hardening agent has also been shown to possess fat reduction qualities. In a roundabout way most anabolic androgenic steroids possess this trait to a degree; by increasing lean tissue we inevitably decrease the total percentage of body fat we carry. Even so, steroids like Trenbolone have been shown to actually reduce stored body fat; while increasing lean tissue, which leads to more pleasing total of fat versus lean tissue, coupled with actual fat reducing qualities, this makes Tren more or less the king of this category when we consider the rates in-which it handles both processes. Beyond Tren, other well-deserving fat loss steroids would include:

While these are the most commonly associated with this principle, other steroids can fill the role as well. For example, one of the absolute best all-around steroids for any purpose is that of Testosterone; were talking about Testosterone of almost any form, i.e. Enanthate, Cypionate, Propionate, Sustanon-250 etc. With increased testosterone levels and adequate diet and exercise we can readily burn more fat than we would otherwise.

Common Mistaken Fat Loss Steroids:

As you may or may not be aware, the performance enhancing world is full of many items that are in-fact not anabolic androgenic steroids; many of these items belong to a host of different classes. Of these commonly mislabeled items the one that stands alone as leader of the pack not only by mistaken identity but in its ability to burn fat as well is that of Human Growth Hormone (HGH.) HGH belongs to a class of hormones known as peptide hormones; these are not steroids. While not a steroid, HGH provides better fat reduction qualities than almost any other performance enhancing drug we can mention. To achieve this purpose use will need to be far extended but the results can be more effective and long lasting if the duration of use is adequate; normally only a low dose is needed to achieve this purpose. Beyond HGH many often labeled fat loss steroids that are not steroids at all include:

  • Clenbuterol
  • Cytomel (t-3)
  • Albuterol
  • Ephedrine

Making Use of Fat Loss Steroids:

A common mistake regarding fat loss steroids and steroids in general for that matter is that if you use them results will happen all on their own. Much of this ridiculous way of thinking is due to the manner in-which steroids are perceived by popular culture; as dark substances from a mystical land of evil. The fact of the matter is, if you are using fat loss steroids, if your diet is not that of a calorie deficiency, meaning you are burning more calories per day than you are taking in, then you wont lose fat. You still have to diet and train/exercise; steroids can simply help you burn more fat than you would otherwise, they are not magical.

 

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EC STACK 30MG/CAPSULE

PRODUCT INFO                                                       Item: 31

 

30mg/capsules eph alternative 200mg/capsule coffeine 100 capsules

 

EC STACK is suitable for the treatment of urinary incontinence in female dogs that are sterilized and whose ovaries and uterus are removed. The urinary incontinence is caused by low pressure in the circular muscles that close the urethra. Enurace is not suitable for the treatment of unwanted urination due to behavioral problems and may only be administered orally. Also contains 200mg Caffeine.

 

EPHEDRINE HCL ALTERNATIVE

Ephedrine’s mechanism of action

Let’s start with ephedrine’s basic mechanism of action. Ephedrine is a sympathomimetic, which means it acts to increase sympathetic activity. Why is this important? Because there is a drop in sympathetic activity when you cut calories. When you get a drop in sympathetic activity you get a decrease in metabolic rate and lower rates of lipolysis (fat mobilization). This can play a significant role in why you plateau on long-term diets. The use of ephedrine and caffeine can stave off this drop in metabolic rate and allow fat loss to continue unabated for several additional weeks.

People often want to compare ephedrine to Clenbuterol. This is fine as long as you realize that they act in different ways. Clenbuterol is a specific beta-2 adrenergic agonist. As such, Clenbuterol interacts directly with beta-2 receptors on muscle and fat tissue. Ephedrine, on the other hand, is not a direct beta-2 agonist. In fact, ephedrine is a poor ligand for the beta-2 receptor. Instead, ephedrine stimulates the release of noradrenaline from sympathetic nerve terminals. The noradrenaline then goes on to interact with muscle and fat cells as a nonspecific adrenergic agonist. This simply means that noradrenaline activates beta-2 receptors, but also other beta-receptors as well as alpha-receptors. So to compare ephedrine and Clenbuterol you must take into account their differences.

The differences between ephedrine and Clenbuterol are important. For example, Clenbuterol is a very potent beta-2 agonist. As a result, within 14 to 21 days, the beta-2 receptors on muscle and fat cells will be drawn into the cell membrane to reduce their availability to Clenbuterol. Then, if you continue to take it, they will be disassembled all together, leaving you insensitive to Clenbuterol. It then takes at least 2-3 weeks for receptors to replenish themselves on muscle and fat cells. Once again, ephedrine is different with respect to down regulation. Ephedrine, being a much weaker agonist, does not cause rapid desensitization and/or down-regulation of adrenergic receptors. This allows ephedrine to be used effectively for many months. Despite these differences, both drugs should be tapered over the course of several weeks before discontinuing them. If you don’t, you will quickly replace the fat you just lost due to an increased appetite and a decreased ability of fat cells to mobilize stored fat.

Choosing the right diet to use with Ephedrine/Caffeine

Your diet will also make a difference in what effects ephedrine will produce. Insulin prevents lipolysis. Insulin has been shown to inhibit lipolysis in a phosphatidylinositol 3-kinase- (PI3K) and phosphodiesterase 3B (PDE3)-dependent manner and to stimulate fractional re-esterification of free fatty acids (FFAs), resulting in inhibition of FFA release (1,2). Even in the presence of ephedrine and/or noradrenaline, a high carbohydrate diet will reduce the fat mobilizing effects of ephedrine. This being the case, I generally recommend a low carb diet to get the most out of ephedrine and caffeine. Keep in mind, however, that over time, your body will not respond so well to ephedrine when on a low carb diet. This should not surprise you considering your metabolic rate (reflecting CNS activity) will drop like a led zeppelin on a low carb diet anyway. So, a periodic carb up is helpful in keeping ephedrine’s thermogenic properties high. A carb up should last no longer than 2 days. I may be helpful to keep fat low during these two days, or if you must eat fat, make sure it is n-3 and/or n-6 fatty acids.

How to use ephedrine to keep that hard earned muscle

You may have heard the Clenbuterol prevents muscle loss during a diet. This is true. Beta-adrenergic stimulation can preserve muscle protein. If you ask your average bodybuilder they will usually tell you that indeed Clenbuterol saves muscle, but ephedrine won’t. They are wrong, of course. In studies measuring body composition as well as weight loss, ephedrine has shown the ability to prevent lean tissue loss (3). In a small double blind study lasting eight weeks, two groups of obese women were given either 20 mg ephedrine with 200 mg caffeine (E+C) or placebo (P) three times daily. After eight weeks weight loss was not significantly different between the groups, but the E + C group lost 4.5 kg more body fat and 2.8 kg less fat free mass (FFM). That is a difference of more than six pounds in eight weeks. The expected decrease in 24-hour energy expenditure (EE) seen in the P group was 10% at day 1 and 13% at day 56, but was only 7% and 8% in the treated group. The higher EE in the E +C group was entirely covered by fat oxidation.

It’s no secret that Clenbuterol has potent anabolic effects in animals when used in dosages around 4 mg per kg body weight. What does seem to be a secret is that this effect is dependent on Clenbuterol’s long and steady activation of the beta receptors (4). The half-life of Clenbuterol is between 34-35 hours. All of the other beta agonists that have been labeled ineffective at preserving or even building muscle have half-lives much shorter, around only 6 hours. It has been demonstrated however, that when you infuse beta agonists with short half-lives, you can elicit the same anabolic activity (4). The reason is because infusion circumvents the half-life issue all together, providing a steady stream of agonist for the beta-receptors.

So what does all this have to do with ephedrine? Well, ephedrine also has a very short half-life, only about 3-4 hours. Keep in mind that this does not mean that ephedrine levels are high for the full 3-4 hours. In reality, they will peak early and then begin to decline quite rapidly. So if you are taking ephedrine every 4 hours, levels are pretty low for most of that time. What’s the solution? Simple. Take ephedrine at least every 2 hours. You will find in doing so that you don’t get the hills and valleys in your mood either. 

NOTE: if you are taking 20 mg ephedrine with 200 mg caffeine every 4 hours, you should half the dose to take it every 2 hours. That way you are still taking the same total amount over the course of the day. You will be surprised at how such a small change can dramatically improve the experience and results of using ephedrine and caffeine.

Points to take home

  • Keep carbs low while using ephedrine. Insulin counter-acts the effects of ephedrine and will ultimately hinder fat loss if carbs remain too high. This does not mean you must go on a ketogenic diet. If total calories are low enough, carbs become less of an issue. But if you are trying to keep muscle by lowering calories only slightly, the carbs will sabotage ephedrine’s action.
  • Ephedrine and Clenbuterol have two different, yet similar mechanisms of action. Nevertheless, don’t use Clenbuterol and ephedrine at the same time, or even in an alternating fashion. Both drugs work through the beta-adrenergic receptor, however, Clenbuterol will shut down beta-receptors within a few weeks. Taking ephedrine at that time will do little for you. Better to use ephedrine from the start of your diet, only switching to Clenbuterol no further than 3 weeks out from the contest.
  • In order to get the most benefit from ephedrine on muscle loss, adjust your dosing schedule to every 2 hours. This will enhance the duration and consistency of beta-adrenergic activity. Be sure to adjust the amount you take each time in order to maintain the total amount taken over the course of a day. For example, instead of taking 20 mg ephedrine and 200 mg caffeine 3 times daily, take 10 mg ephedrine and 100 mg caffeine 6 times daily.

 

Side Effects

Side Effects are common when this drug is abused, and if they start occurring the user should lower the dosage or frequency of use.

These are the most frequently seen side effects of ephedrine:

  • Heart issues such as hypertension
  • Sweating/flushing
  • Stomach problems including nausea
  • Difficulty urinating
  • Insomnia, confusion, agitation, restlessness
  • Dry mouth, headache, dizziness

 

ECA Stack

The ECA stack is popular in bodybuilding circles as an appetite suppressor and fat burner. The letter “E” stands for ephedrine 25-50mgs, “C” for caffeine 200-300mgs, and “A” for aspirin 100mgs. Among other factors, the precise dosage will greatly depend on body weight.

In general, users should start out with a low dose and then work their way up. Bodybuilders usually split the dosage into 2-3 parts to be taken during the day. This strategy is employed by both males and females.

$80.00  Inc Tax
CLENBUTEROL 200 MG/ML 30 ML

PRODUCT INFO                                            Item: 32

ALTERNATIVE STEROID NAMES:Contraspasmin, Clenasma, Novegam, Contrasmina, Cesbron, Clenbuterol Pharmachim, Spasmo-mucosolvan, Ventolase, Spiropent
ACTIVE SUBSTANCE:Clenbuterol hydrochloride 200mg/ml 30 ml
  
Strength Gains:@
Mass & Weight Gains:@
Fat Burning:@@@@@@
Cutting/Definition:@@@@@@@@@
Side Effects:@@@@
Ability to keep Gains:@
  
Usual dosages:40 - 260 mcg per day
  
Detection time:4 -5 days.
  
Best combined with:
Bulking:HGH (rarely used for bulking).
Cutting:Cytomel (T-3 thyroid), HGH
  
Beginner Use:@@@@@@@@
Female Use:@@@@@@@@@
  
Pros:Very cheap, efective fat burner, low side effects.
Cons:Limited effective time (4-6 weeks), low anabolic effect.

 

Pharmaceutical Name: clenbuterol hydrochloride

Chemical Structure: (RS)-1-(4-amino-3,5-dichlorophenyl)-2-(tert butylamino)ethanol

Effective Dose: 40-260 mcg/day

Clenbuterol is a widely used bronchodilator in many parts of the world. The drug is most often prepared in 40 mcg tablets, but it is also available in syrup and injectable form. Clenbuterol belongs to a broad group of drugs knows as sympathomimetics. These drugs affect that sympathetic nervous system in a wide number of ways, largely mediated by the distribution of adrenoceptors. There are actually nine different types of these receptors in the body, which are classified as either alpha or beta and further subcategorized by type number. Depending on the specific affinities of these agents for the various receptors, they can potentially be used in the treatment of conditions such as asthma, hypertension, cardiovascular shock, arrhythmias, migraine headaches and anaphylactic shock.

The drug clenbuterol is specifically a selective beta-2 sympathomimetic, primarily affecting only one of the three subsets of beta-receptors. Of particular interest is the fact that this drug has little beta-1 stimulating activity. Since beta-1 receptors are closely tied to the cardiac effects of these agents, this allows clenbuterol to reduce reversible airway obstruction (and effect of beta-2 stimulation) with much less cardiovascular side effects compared to nonselective beta agonists. Clinical studies with this drug show it is extremely effective as a bronchodilator, with a low level of user complaints and high patient compliance. Clenbuterol also exhibits an extremely long half-life in the body, which is measured to be approximately 34 hours long. This makes steady blood levels easy to achieve, requiring only a single or twice daily dosing schedule at most. This of course makes it much easier for the patient to use, and may tie in to its high compliance rate. To spite that clenbuterol is available in a wide number of other countries however; this compound has never been approved for use in the United States. The fact that there are a number of similar, effective asthma medications already available in this country may have something to do with this, as a prospective drug firm would likely not find it a profitable enough product to warrant undergoing the expense of the FDA approval process. Regardless, foreign clenbuterol preparations are widely available on the U.S. black market.

In animal studies clenbuterol is shown to exhibit anabolic activity, obviously an attractive trait to the athlete This compound is additionally a known thermogenic, with beta-2 agonists like clenbuterol shown to directly stimulate fat cells and accelerate the breakdown of triglycerides to form free fatty acids. Its efficacy in this area makes clenbuterol a very attractive, and today almost mandatory, pre-contest drug. Those interested in this drug are most often hoping it will impart a little of both benefits, promoting the loss of body fat while imparting strength and muscle mass increases. But as was well pointed out by a review published in the August 1995 issue of Medicine and Science in Sports and Exercise, the possible anabolic activities in humans are very questionable, and based only on animal data using much larger doses than would be required for bronchodilation . With such reports there has been a lot of debate lately as to whether or not clenbuterol is really anabolic at all. Some seem to swear by the fact that it builds muscle regardless, firmly sticking by "clen" as a great off-season or adjunct anabolic. To others such reports are confirmation that athletes have wasted valuable time and money on drugs that do not work as they are intended to by the user.

This debate continues today, with many still using clenbuterol as a potential anabolic. With this in mind athletes will tailor their dosage and cycling of this product individually depending on which of the two "possible" results are more desired, and how much side effects are to be tolerated. The possible side effects of clenbuterol include those of other CNS stimulants, and include such occurrences as shaky hands, insomnia, sweating, increased blood pressure and nausea. These side effects will generally subside after a week or so of use however, once the user becomes accustomed to the drug. One would typically start a cycle by gradually increasing the dosage each day until a desired range is established. This process will minimize the unwanted side effects seen from the drug; which otherwise might be dramatic if a large dose is administered from the onset. Men generally end up in the range of 2-8 tablets per day, although some people do claim to tolerate even higher dosages. Women get by on less, generally 2-4 tablets daily. Very quickly, the drug will elevate the body temperature. The rise is not usually dramatic, perhaps a half of a degree or so, sometimes a little more. This elevation is due to your body burning excess energy (largely from fat) and is usually not uncomfortable.

Now that it is working, the number of consecutive days clenbuterol can be used is believed to be dependent on the goal of the individual. To be clear, the athletic benefits of this drug will only last for a limited time and then diminish, largely due to beta-receptor down regulation. When using it for fat loss, the primary effect of the drug, it seems to work well for approximately 4-6 weeks. During this period, users will want to constantly monitor their body temperature. We are assured clenbuterol is working by the temperature elevation. Once the temperature drops back to normal, clenbuterol is no longer exhibiting a thermogenic effect. At this point increasing the dosage would not be very effective, and a break for at least a few weeks should be taken before it is used again effectively. If one is looking for strength gains, clenbuterol appears to be effective for a much shorter period of time, around 3-4 weeks. This may be due to an absence of real anabolic effect, with the strength gain seen with clenbuterol possibly due only to the stimulant properties of the drug (similar to the strength boost seen by ephedrine users). Again however, this is still debated.

Many competitors also find the fat burning effect of clenbuterol can be further enhanced by additional substances. When combined with thyroid hormones, specifically the powerful Cytomel, the thermogenic effect can become extremely dramatic. This can be to a point that the athlete could shred exceptional amounts of extra fat during contest preparations, without a dramatic restriction in calories. Such a mix can be further used during a steroid cycle, eliciting a much harder look from the anabolics. These cutting agents can often greatly inhibit extra fat storage during the cycle, even when using strong aromatizing androgens. A clenbuterol/thyroid mix is also common when using growth hormone, further enhancing the thermogenic and anabolic effect of this therapy.

$20.00
$25.00
SIBUFIT 20 MG/ 30 CAPSULE

PRODUCT INFO                                                              Item: 30

 

20mg/capsule 30 capsules

What is sibutramine?

Sibutramine was withdrawn from the U.S. market in October 2010.

Sibutramine affects chemicals in the brain that affect weight maintenance.

Sibutramine is used together with diet and exercise to treat obesity that may be related to diabetes, high cholesterol, or high blood pressure.

Sibutramine may also be used for other purposes not listed in this medication guide.

What is the most important information I should know about sibutramine?

Sibutramine was withdrawn from the U.S. market in October 2010.

Do not use sibutramine if you have taken an MAO inhibitor such as furazolidone (Furoxone), isocarboxazid (Marplan), phenelzine (Nardil), rasagiline (Azilect), selegiline (Eldepryl, Emsam), or tranylcypromine (Parnate) in the last 14 days. Serious, life threatening side effects can occur if you use sibutramine before the MAO inhibitor has cleared from your body.

You should not take sibutramine if you are allergic to it, or if you have severe or uncontrolled high blood pressure, an eating disorder (anorexia or bulimia), if you are taking stimulant diet pills, or if you have a history of coronary artery disease, stroke, or heart disease.

Before taking sibutramine, tell your doctor if you have glaucoma, high blood pressure, liver or kidney disease, depression, underactive thyroid, seizures, a bleeding disorder, a history of gallstones, or if you are older than 65 or younger than 16.

Tell your doctor about all prescription and over-the-counter medications you use, especially antidepressants, cold or allergy medication, narcotic pain medicine, or migraine headache medicines.

Tell your doctor if you do not lose at least 4 pounds after taking the medication for 4 weeks along with a low calorie diet.

What should I discuss with my healthcare provider before taking sibutramine?

Do not use sibutramine if you have taken an MAO inhibitor such as furazolidone (Furoxone), isocarboxazid (Marplan), phenelzine (Nardil), rasagiline (Azilect), selegiline (Eldepryl, Emsam), or tranylcypromine (Parnate) in the last 14 days. Serious, life threatening side effects can occur if you use sibutramine before the MAO inhibitor has cleared from your body.

You should not take this medication if you are allergic to sibutramine, or if you have:

  • severe or uncontrolled hypertension (high blood pressure);

  • an eating disorder (anorexia or bulimia);

  • a history of coronary artery disease (atherosclerosis);

  • a history of heart disease (congestive heart failure, heart rhythm disorder);

  • a history of heart attack or stroke; or

  • if you are taking stimulant diet pills.

If you have any of these other conditions, you may need a sibutramine dose adjustment or special tests:

  • glaucoma;

  • high blood pressure;

  • liver disease;

  • kidney disease;

  • depression;

  • underactive thyroid;

  • epilepsy or seizure disorder;

  • a bleeding or blood clotting disorder;

  • a history of gallstones; or

  • if you are older than 65 or younger than 16.

FDA pregnancy category C. It is not known whether sibutramine will harm an unborn baby. Tell your doctor if you are pregnant or plan to become pregnant while using sibutramine.

It is not known whether sibutramine passes into breast milk or if it could harm a nursing baby. Do not use this medication without telling your doctor if you are breast-feeding a baby.

Do not give this medication to anyone younger than 16 years old.

How should I take sibutramine?

Take exactly as prescribed by your doctor. Do not take in larger or smaller amounts or for longer than recommended. Follow the directions on your prescription label.

Sibutramine is usually taken once daily. Follow your doctor's instructions.

Your doctor may occasionally change your dose to make sure you get the best results.

Sibutramine can be taken with or without food.

You should lose at least 4 pounds during the first 4 weeks of taking sibutramine and eating a low calorie diet. Tell your doctor if you do not lose at least 4 pounds after taking the medication for 4 weeks.

Your blood pressure and pulse will need to be checked often. Visit your doctor regularly.

Sibutramine should not be taken for longer than 2 years.

Store at room temperature away from moisture, heat, and light.

Do not share sibutramine with another person. Keep the medication in a place where others cannot get to it.

What happens if I miss a dose?

Take the missed dose as soon as you remember. Skip the missed dose if it is almost time for your next scheduled dose. Do not take extra medicine to make up the missed dose.

What happens if I overdose?

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include headache, dizziness, and fast heart rate.

What should I avoid while taking sibutramine?

Sibutramine may impair your thinking or reactions. Be careful if you drive or do anything that requires you to be alert.

Do not take any other prescription or over-the-counter weight-loss products without your doctor's advice.

Avoid taking cough and cold or allergy medications while taking sibutramine.

Avoid drinking alcohol while taking sibutramine.

Sibutramine side effects

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficult breathing; swelling of your face, lips, tongue, or throat.

Stop using sibutramine and call your doctor at once if you have a serious side effect such as:

  • fast, pounding, or uneven heartbeats;

  • new or worsening shortness of breath;

  • agitation, hallucinations, fever, tremor, overactive reflexes, nausea, vomiting, diarrhea, loss of coordination, dilated pupils;

  • very stiff (rigid) muscles, high fever, sweating, confusion, feeling like you might pass out;

  • easy bruising or bleeding (nosebleeds, bleeding gums, or any bleeding that will not stop);

  • dangerously high blood pressure (severe headache, blurred vision, buzzing in your ears, anxiety, seizure);

  • chest pain or heavy feeling, pain spreading to the arm or shoulder, general ill feeling; or

  • sudden numbness or weakness (especially on one side of the body), problems with vision, speech, or balance.

Less serious side effects may include:

  • dry mouth, upset stomach;

  • changes in appetite;

  • constipation, stomach pain;

  • headache, back pain, joint pain;

  • feeling nervous, dizzy, or depressed;

  • flu symptoms, runny or stuffy nose, sore throat, cough;

  • warmth, redness, or tingly feeling under your skin;

  • trouble sleeping (insomnia); or mild skin rash.

Sibutramine Dosing Information

Usual Adult Dose for Obesity:

Sibutramine was voluntarily withdrawn from the US market by the manufacturer in October, 2010 due to clinical trial data indicating an increased risk of heart attack and stroke. The following dosage information applies to when the drug was available in the US.

Initial Dose: 10 mg orally once a day.

Usual Geriatric Dose for Obesity:

Sibutramine was voluntarily withdrawn from the US market by the manufacturer in October, 2010 due to clinical trial data indicating an increased risk of heart attack and stroke. The following dosage information applies to when the drug was available in the US.

Clinical studies of sibutramine did not include sufficient numbers of patients over 65 years of age. Sibutramine is contraindicated in this group of patients.

Usual Pediatric Dose for Obesity:

Sibutramine was voluntarily withdrawn from the US market by the manufacturer in October, 2010 due to clinical trial data indicating an increased risk of heart attack and stroke. The following dosage information applies to when the drug was available in the US.

Based on one study (n=60)

14 to 17 years:
Initial Dose: 10 mg orally once a day.

What other drugs will affect sibutramine?

Before using sibutramine, tell your doctor if you regularly use other medicines that make you sleepy (such as cold or allergy medicine, sedatives, narcotic pain medicine, sleeping pills, muscle relaxers, and medicine for seizures, depression, or anxiety). They can add to sleepiness caused by sibutramine.

Ask your doctor before taking any decongestants, cough medicine, or other diet pills.

The following drugs can interact with sibutramine. Tell your doctor if you are using any of these:

  • lithium (Lithobid, Eskalith, and others);

  • tryptophan or L-tryptophan;

  • ketoconazole (Nizoral);

  • an antibiotic such as erythromycin (E.E.S., EryPed, Ery-Tab, Erythrocin);

  • an antidepressant such as citalopram (Celexa), desvenlafaxine (Pristiq), duloxetine (Cymbalta), fluoxetine (Prozac, Sarafem, Symbyax), paroxetine (Paxil), sertraline (Zoloft), venlafaxine (Effexor), and others;

  • ergot medicine such as dihydroergotamine (D.H.E. 45, Migranal Nasal Spray), ergonovine (Ergotrate), ergotamine (Ergomar), or methylergonovine (Methergine).

  • migraine headache medicine such as sumatriptan (Imitrex) or zolmitriptan (Zomig); or

  • narcotic pain medication such as fentanyl (Actiq, Duragesic, Fentora, Onsolis), meperidine (Demerol), pentazocine (Talwin).

This list is not complete and other drugs may interact with sibutramine. Tell your doctor about all medications you use. This includes prescription, over the counter, vitamin, and herbal products. Do not start a new medication without telling your doctor.

$20.00
$30.00
CYTOMEL T3 100 MG/ML 30 ML

PRODUCT INFO                                            Item: 33

ALTERNATIVE STEROID NAMES:Tertroxin, Cytomel, Ro-Thyronine, Cytomel tabs, Sanabo, Tertroxin, Linomel, Thyrotardin, Tiromel
ACTIVE SUBSTANCE:Liothyronine Sodium 100 mg/ml 30 ml
  
Strength Gains: 
Mass & Weight Gains: 
Fat Burning:@@@@@@@@
Cutting/Definition:@@@@@@@@@
Side Effects:@@@@@@@@
Ability to keep Gains:@
  
Usual dosages:25 - 100 mcg per day
  
Detection time:NA
  
Best combined with:
Bulking:HGH (rarely used for bulking).
Cutting:Clenbuterol, HGH
  
Beginner Use:@@@@@@
Female Use:@@@@@@@@
  
Pros:Cheap, effective fat burner.
Cons:Potentially dangerous when used for a long time.

 

Pharmaceutical Name: liothyronine sodium 

Chemical Structure: tri-iodio-thyronine (T3) 

Molecular weight of base: 650.9776 

Effective Dose: 25-100 mcg / day orally 

 

What is liothyronine?

Liothyronine is a man-made form of a hormone that is normally produced by your thyroid gland to regulate the body's energy and metabolism. Liothyronine is given when the thyroid does not produce enough of this hormone on its own.

Liothyronine treats hypothyroidism (low thyroid hormone). Liothyronine is also used to treat or prevent goiter (enlarged thyroid gland), and is also given as part of a medical tests for thyroid disorders.

Liothyronine should not be used to treat obesity or weight problems.

Liothyronine may also be used for purposes not listed in this medication guide.

What is the most important information I should know about liothyronine?

Since thyroid hormone occurs naturally in the body, almost anyone can take liothyronine. However, you may not be able to use this medication if you have a thyroid disorder called thyrotoxicosis, or an adrenal gland problem that is not controlled by treatment.

Before taking liothyronine, tell your doctor if you have heart disease, angina (chest pain), coronary artery disease, congestive heart failure, diabetes, or problems with your pituitary or adrenal glands.

Tell your doctor if you are pregnant or breast-feeding.

To be sure this medication is helping your condition, your blood may need to be tested often. Visit your doctor regularly.

Keep using this medicine as directed, even if you feel well. You may need to take thyroid medication for the rest of your life.

Call your doctor if you notice any signs of thyroid toxicity, such as chest pain, fast or pounding heartbeats, feeling hot or nervous, or sweating more than usual.

What should I discuss with my healthcare provider before taking liothyronine?

Since thyroid hormone occurs naturally in the body, almost anyone can take liothyronine. However, you may not be able to use this medication if you have a thyroid disorder called thyrotoxicosis, or an adrenal gland problem that is not controlled by treatment.

To make sure you can safely take liothyronine, tell your doctor if you have any of these other conditions:

  • heart disease, angina (chest pain);

  • coronary artery disease;

  • congestive heart failure;

  • any type of diabetes; or

  • problems with your pituitary or adrenal gland.

FDA pregnancy category A. Liothyronine is not expected to harm an unborn baby. However, tell your doctor if you become pregnant, since your dose needs may be different during pregnancy.

Small amounts of liothyronine can pass into breast milk, but this is not expected to harm a nursing baby. However, do not use this medication without telling your doctor if you are breast-feeding a baby.

How should I take liothyronine?

Take exactly as prescribed by your doctor. Do not take in larger or smaller amounts or for longer than recommended. Follow the directions on your prescription label.

Liothyronine is usually taken once daily. Follow your doctor's instructions.

Your doctor may occasionally change your dose to make sure you get the best results.

If you are switching to liothyronine from any other thyroid medication, stop using the other medication before you start taking liothyronine.

To be sure this medication is helping your condition, your blood may need to be tested often. Visit your doctor regularly.

Keep using this medicine as directed, even if you feel well. You may need to take thyroid medication for the rest of your life.

Call your doctor if you notice any signs of thyroid toxicity, such as chest pain, fast or pounding heartbeats, feeling hot or nervous, or sweating more than usual.

If you need surgery, tell the surgeon ahead of time that you are using liothyronine. You may need to stop using the medicine for a short time.

Store at room temperature away from moisture and heat.

What happens if I miss a dose?

Take the missed dose as soon as you remember. Skip the missed dose if it is almost time for your next scheduled dose. Do not take extra medicine to make up the missed dose.

What happens if I overdose?

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include headache, sweating, diarrhea, irregular menstrual periods, confusion, weakness, swelling in your hands or feet, fast heart rate, chest pain, feeling short of breath, fainting, or feeling nervous, restless, or irritable.

What should I avoid while taking liothyronine?

Do not change brands or change to a generic product without first asking your doctor. Different brands of liothyronine may not work the same. If you get a prescription refill and your new pills look different, talk with your pharmacist or doctor.

If you also take cholestyramine (Prevalite, Questran) or colestipol (Colestid), avoid taking these medications within 4 hours before or after you take liothyronine.

Liothyronine side effects

Get emergency medical help if you have any of these signs of an allergic reaction: hives; difficult breathing; swelling of your face, lips, tongue, or throat.

Less serious side effects may include temporary hair loss (especially in children).

Liothyronine Dosing Information

Usual Adult Dose for Hypothyroidism:

Initial dose: 25 mcg orally once a day; may be increased by up to 25 mcg every 1 to 2 weeks depending on the patient's clinical response and laboratory findings

Maintenance dose: 25 to 75 mcg daily

Usual Adult Dose for Myxedema:

Initial dose: 5 mcg orally once a day; may be increased by 5 to 10 mcg every 1 to 2 weeks depending on the patient's clinical response and laboratory findings

When treatment is up to 25 mcg/day, the dosage may be increased by 5 to 25 mcg every week or two until a satisfactory therapeutic response is attained.

Maintenance dose: 50 to 100 mcg daily

Usual Adult Dose for Myxedema Coma:

25 to 50 mcg intravenously

If the patient has known or suspected cardiovascular disease, then an initial dose of 10 to 20 mcg in conjunction with cardiac monitoring is recommended.

Subsequent doses should be determined by the patient's clinical condition and response to treatment. Generally, doses should be administered at least 4 hours and no more than 12 hours apart. Caution is advised in adjusting the dose due to the potential of acute and large changes to precipitate adverse cardiovascular events.

Available clinical data indicate reduced mortality in patients receiving at least 65 mcg/day in the initial days of treatment. However, there is limited clinical experience with dosages above 100 mcg/day.

Oral thyroid hormone therapy should be substituted as soon as the patient is stable and able to take oral medication. If levothyroxine rather than liothyronine is used, the prescriber should bear in mind that there is a delay of several days in the onset of levothyroxine activity and that intravenous therapy should be discontinued gradually.

Usual Adult Dose for Thyroid Suppression Test:

75 to 100 mcg orally once a day for 7 days

Radioactive (131) iodine uptake should be determined before and after the administration of thyroid hormone. If thyroid function is under normal control, the radioactive iodine uptake should drop significantly after treatment. A 50% or greater suppression of uptake indicates a normal thyroid-pituitary axis and rules out thyroid gland autonomy.

Liothyronine should be administered cautiously if there is a strong suspicion of thyroid gland autonomy, as exogenous hormone effects will be additive to those of the endogenous source.

Usual Adult Dose for TSH Suppression:

Initial dose: 5 mcg orally once a day; may be increased by 5 to 10 mcg every 1 to 2 weeks depending on the patient's clinical response and laboratory findings

When treatment is up to 25 mcg/day, the dosage may be increased by 12.5 to 25 mcg every week or two until a satisfactory therapeutic response is attained.

Maintenance dose: 75 mcg daily

Usual Pediatric Dose for Hypothyroidism:

Congenital hypothyroidism/Hypothyroidism:
Infants and Children less than or equal to 3 years:
Initial dose: 5 mcg orally once a day; may be increased by 5 mcg every 3 days to a maximum dosage of 20 mcg/day for infants and 50 mcg/day for children 1 to 3 years of age.

Hypothyroidism: 
Initial dose: Children: 5 mcg orally once a day. Increase in 5 mcg/day increments every 3 to 4 days
Usual maintenance dose: 
Infants: 20 mcg orally once a day
Children 1 to 3 years: 50 mcg orally once a day
Children greater than 3 years: Full adult dosage may be necessary 

Goiter (nontoxic): 
Children:
Initial dose: 5 mcg orally once a day. Increase in 5 mcg/day increments every 1 to 2 weeks
Maintenance dose: 15 to 20 mcg orally once a day

Goiter (nontoxic): 
Children: 5 mcg orally once a day. Increase in 5 mcg/day increments every 1 to 2 weeks
Maintenance dose 15 to 20 mcg orally once a day

What other drugs will affect liothyronine?

Tell your doctor about all other medicines you use, especially:

  • birth control pills or hormone replacement therapy;

  • a blood thinner such as warfarin (Coumadin);

  • digoxin (digitalis, Lanoxin);

  • epinephrine (EpiPen) or norepinephrine (Levophed);

  • insulin or oral diabetes medication;

  • medications that contain iodine (such as I-131);

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